During pregnancy in the body of a woman, a temporary and vital organ for the successful gestation and development of the fetus, called the placenta, is formed. The process of formation of the placenta is completed by the sixteenth week of pregnancy with its normal course. This organ is formed as a result of implantation of a fertilized egg into the uterus. The main functions of the placenta is to provide the fetus with the right amount of nutrients and transport oxygen to it. With the help of ultrasound can determine the thickness of the placenta, which in turn will show how it copes with its duties.
Causes of placental hyperplasia
The normal thickness of the placenta is about as much as the current gestation period. The increase in normal growth of the placenta reaches its peak by the thirty-seventh week and becomes 33.75 mm.
Further, the growth of the placenta ceases and at the onset of labor may slightly decrease to 33.25 mm. Thickening of the embryonic organ and its inconsistency with the norms of development is called placental hyperplasia, which in turn will talk about the presence in the body of certain pathologies.
Causes of thickening of the placenta:
- acute viral infections;
- a decrease in hemoglobin in the blood to less than 80 g / l, due to the development of severe anemia;
- chronic sexual infections (ureaplasma, toxoplasma, syphilis, mycoplasma, chlamydia, gonorrhea);
- different rhesus blood from the mother and child and conflict in this case;
- late gestosis.
Often doctors can not find the reason for the thickening of the "child's place."
If the thickness of the placenta is increased in the early stages, then this can only be determined by means of ultrasound, Its pathological increase proceeds without any symptoms.
At later stages, the fetal movements may change (from too violent to extremely slow movements), and during the use of cardiotocography, a change in the fetal heartbeat can be detected.
Diagnostic measures are focused on identifying the causes of placental hyperplasia. For this future mother, the following actions are assigned:
- biochemical blood test;
- cardiogram fetus;
- general tests of urine and blood;
- a screening for the detection of sexually transmitted infections and TORCH infections;
- blood test for antibodies;
- UZI Dopplera;
- gynecological smear on the flora.
Treatment of placental hyperplasia
- If, during additional diagnostics, it is found that along with the thickening of the placenta, the intrauterine development of the child is delayed, then the future mother must be hospitalized and further treatment should be performed in the hospital.
- To normalize the thickness of the placenta may be used drugs that improve the microcirculation of the embryonic organ, as well as medicines that dilute blood.
- The most important stage of treatment is taking drugs to improve the oxygenation of the fetus and the placenta itself.
- To prevent the destruction of cells, the most effective way is to use essential phospholipids, which perfectly perform their construction function.
- Also, the treatment of placental hyperplasia can include vitamin therapy, antiviral therapy, treatment of late gestosis, intrauterine blood transfusion, normalization of blood sugar levels.
- If the placenta increase is insignificant, and the fetus develops according to the schedule and norms, then the treatment of hyperplasia may not be necessary.
Consequences and prevention of placental hyperplasia
Thickening of the placenta provokes the development of fetoplacental insufficiency, in which the supply of nutrients and oxygen to the baby fails, resulting in a slowdown in its intrauterine development and a greater likelihood of complication of the birth process.
To avoid such consequences, it is necessary to timely pass all the diagnostic measures prescribed in the female consultation.